Discovery of GS-9973, a selective and orally efficacious inhibitor of spleen tyrosine kinase.

نویسندگان

  • Kevin S Currie
  • Jeffrey E Kropf
  • Tony Lee
  • Peter Blomgren
  • Jianjun Xu
  • Zhongdong Zhao
  • Steve Gallion
  • J Andrew Whitney
  • Deborah Maclin
  • Eric B Lansdon
  • Patricia Maciejewski
  • Ann Marie Rossi
  • Hong Rong
  • Jennifer Macaluso
  • James Barbosa
  • Julie A Di Paolo
  • Scott A Mitchell
چکیده

Spleen tyrosine kinase (Syk) is an attractive drug target in autoimmune, inflammatory, and oncology disease indications. The most advanced Syk inhibitor, R406, 1 (or its prodrug form fostamatinib, 2), has shown efficacy in multiple therapeutic indications, but its clinical progress has been hampered by dose-limiting adverse effects that have been attributed, at least in part, to the off-target activities of 1. It is expected that a more selective Syk inhibitor would provide a greater therapeutic window. Herein we report the discovery and optimization of a novel series of imidazo[1,2-a]pyrazine Syk inhibitors. This work culminated in the identification of GS-9973, 68, a highly selective and orally efficacious Syk inhibitor which is currently undergoing clinical evaluation for autoimmune and oncology indications.

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عنوان ژورنال:
  • Journal of medicinal chemistry

دوره 57 9  شماره 

صفحات  -

تاریخ انتشار 2014